Monday, January 9, 2012

The Healing Powers of Vinegar


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No single food or supplement can prevent cancer, amazingly however, vinegar
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Your Hairdresser Can Help Catch Cancer







Your Hairdresser Can Help Catch Cancer



Make a list of your most intimate relationships and, in all
probability, your hairdresser or barber should be on it. You’ve probably
swapped many stories during washings and trimmings, and maybe you’ve
even traded advice about life and love. (And, really, how many other
people run their fingers through your hair?!)





Now, according to the Harvard School of Public Health in Boston, there’s even more that your hairdresser can and should do -- alert you about possible skin cancer.



Since the only thing that my hairdresser has ever alerted me about
has been which shampoo would add the most volume to my hair, I called
Alan Geller, MPH, RN, senior lecturer in the school’s department of
society, human development and health, because he recently led a study
on the topic.



A BIRD’S-EYE VIEW




Who else sees -- really sees -- your scalp and the back of your neck?
Understanding that unique view, Geller and his colleagues wondered if
hair professionals use, or could use, that perspective to help look for
skin cancers. If so, it could save lives, because while melanomas of the
scalp and neck represent only about 6% of all melanomas in the US, they
account for about 10% of all melanoma deaths. Geller told me that one
reason scalp and neck cancers are such high-risk cancers is that we
can’t easily see those areas of the body, so cancers there tend to elude
early detection -- and the later a cancer is detected, the greater the
risk for death.

HOW IN-THE-KNOW ARE HAIRDRESSERS?




The researchers analyzed surveys completed by 203 male and female
hairdressers and barbers from 17 salons in the Houston area. There were
43 questions total, including questions about knowledge of the ABCD rule
of detecting cancerous lesions (asymmetry-border-color-diameter),
personal practices in caring for their own skin (such as wearing
sunscreen and hats) and health communication practices.

What researchers discovered: Although fewer than one-third
of these professionals had had any formal training in skin cancer
detection, many of them seemed to understand the essential elements --
90% agreed that a customer should see a doctor for a mole that’s
changing in size or that bleeds... 89% for a mole that’s changing
color... and 78% for a mole that itches. More than one-third (37%) said
that they had actually inspected the scalps of more than half of their
customers in the past month, and 29% said that they had examined the
necks of more than half of their customers in the past month.
Researchers were also happily surprised to find that 58% of the
hairdressers had already recommended that a customer see a doctor about
an abnormal mole that they had found. In addition, Geller was encouraged
by the fact that most respondents recognized the importance of their
role -- half said that they would like to receive formal skin cancer
education, and 69% said that they would give customers a skin cancer
pamphlet if they had one.
WHY TALKING TO YOUR HAIRDRESSER IS KEY



Most people see their physician about twice a year, and only 15% of
people see a dermatologist annually. But most people see hairdressers or
barbers much more often, so their observations could be extremely
valuable, said Geller. Right now, there are no cosmetology rules or
incentives that encourage hair professionals across the US to perform
skin cancer screenings on customers, but Geller envisions a future where
hair professionals are on the front line of skin cancer sightings,
alerting customers about suspicious skin lesions and recommending that
they see their doctors.

The Harvard group isn’t stopping there. Geller’s colleague,
researcher Elizabeth Bailey, MD, is working with the Melanoma Foundation
of New England on a 20-minute pilot program to educate hair
professionals about checking customers for scalp and neck skin cancers. I
asked the foundation’s executive director, Deb Girard, whether Daily Health News could see the brochure that she and Dr. Bailey have developed. She was delighted to share it -- you can see it at http://www.mfne.org/prevent-melanoma/the-skinny-on-skin. You’re welcome to download it for free and share it with your own hair professional.

Next on the agenda: Massage therapists, whose work gives
them the chance to look closely at other parts of their customers’ skin.
Geller reminded me that skin cancer is the only visible cancer and
repeated a saying that he loves, "Melanoma writes its message on the
skin for all of us to see." He wants more people to be able to read that
message.
Source(s): Alan
C. Geller, MPH, RN, senior lecturer, department of society, human
development, and health, Harvard School of Public Health, Boston.

Deb Girard, executive director, Melanoma Foundation of New England,
Concord, Massachusetts.







Friday, January 6, 2012

Match Meals in Minutes for People with Diabetes (American Diabetes Association).

The Perfect Panini
Special from Bottom Line/Personal November 1, 2011-->Crisp crust, warm tasty fillings with melted cheese—no wonder paninis have become so popular. Ciabatta bread typically is used, but a large baguette, Italian sub roll or focaccia works as well.Paninis are pressed down lightly while cooking, but you don’t need a special panini press. Simply place the sandwich in a skillet, and press it down with a lid that is smaller than the skillet. Or place another skillet over the panini, and add a large can or other weight on top.TURKEY AND CHEESE PANINI1 ciabatta bread (about 8 ounces)2 Tablespoons olive oil2 medium garlic cloves, minced½ pound sliced low-sodium smoked turkey breast½ cup sliced, drained canned roasted red peppers½ cup crumbled goat cheese10 to 12 fresh basil leavesOlive oil sprayCut bread into two five-inch pieces, and slice them open lengthwise. Spread olive oil on the top halves, and sprinkle garlic over the oil. Place turkey on the bottom halves. Place roasted red peppers over the turkey. Sprinkle goat cheese over the red peppers, and top each sandwich with five or six basil leaves. Cover with the top halves. Heat a large, nonstick skillet over medium heat, and spray with olive oil spray. Add the paninis, and press down with a lid. Cook for three minutes. Turn them over, and press with a lid. Cook another three minutes. Serves two.FLORENTINE CHICKEN PANINI1 ciabatta bread (about 8 ounces)Olive oil spray½ cup sliced red onion1 garlic clove, minced2 Tablespoons mayonnaise6 ounces cooked chicken strips2 ounces sliced provolone cheese½ cup fresh spinach leaves, chopped or cookedCut bread into two five-inch pieces, and slice them open lengthwise. Heat a large, nonstick skillet over medium heat, and spray with olive oil spray. Sauté the onion and garlic about three minutes. Remove and mix the garlic and onion into the mayonnaise. Spread the top halves with the mayonnaise. Top the bottom halves with the chicken, and place the provolone over the chicken. Place the spinach over the cheese. Cover with the top halves. Heat the skillet again, and spray with olive oil spray. Add the paninis, and press down with a lid. Cook for three minutes. Turn them over, and press with a lid. Cook another three minutes. Serves two.ROAST BEEF AND ARUGULA PANINI1 ciabatta bread (about 8 ounces)3 Tablespoons mayonnaise6 ounces sliced deli roast beef1 cup sliced ripe pear2 ounces Brie cheese, cut into half-inch strips1 cup arugula leavesOlive oil sprayCut bread into two five-inch pieces, and slice them open lengthwise. Spread the top halves with mayonnaise. Put the roast beef on the two bottom halves. Place pear slices over the roast beef, and arrange the Brie cheese over the pear slices. Top with the arugula leaves. Cover with the top halves of the bread. Heat a large, nonstick skillet over medium heat. Spray with olive oil spray. Add the paninis, and press down with a lid. Cook three minutes. Turn them over, and press with a lid. Cook another three minutes. Serves two. Linda Gassenheimer is an award-winning author of several cookbooks, most recently, The Flavors of the Florida Keys (Atlantic Monthly) and Mix ‘n’ Match Meals in Minutes for People with Diabetes (American Diabetes Association). She writes the syndicated newspaper column “Dinner in Minutes.” www.DinnerInMinutes.comRate this listing0 vote

Thursday, January 5, 2012

IF YOU WANT TO LIVE A LONG TIME, LOVE AND BE LOVED"

Like Alzheimer’s disease, frontotemporal dementia involves symptoms such as memory loss

Will You Get This Kind of Dementia? Consumers of health care are increasingly sophisticated (especially you readers of Daily Health News!) and now pretty well-versed in the differing symptoms of related diseases -- such as different types of cancer and different cardiovascular problems. Dementia, on the other hand, tends to be thought of as one problem that encompasses lots of symptoms involving forgetfulness and fuzzy thinking -- but, in fact, it too takes many forms with distinct differences among them. Now scientists have just learned something important about one kind, called frontotemporal dementia (FTD), that accounts for somewhere between 2% and 10% of all dementia cases. Like Alzheimer’s disease, frontotemporal dementia involves symptoms such as memory loss and cognitive decline, but the causes of the two types of dementia are different. And, I was told by Stephen M. Strittmatter, MD, PhD, the Vincent Coates Professor of Neurology at Yale School of Medicine, the more we understand about the differences between the two, the closer we come to finding treatments for them. A Different Form of DementiaWhile Alzheimer’s is characterized by the buildup of amyloid plaques in the brain, frontotemporal dementia is triggered by the degeneration of nerve cells in the frontal and temporal lobes, the areas involved with language, behavior and personality. Some individuals with this condition undergo personality changes (such as obnoxious or even obscene behavior, apathy, poor judgment) and act in socially inappropriate ways. Other important differences: Frontotemporal dementia is more likely to strike at a younger age -- age 40 to 70 -- than Alzheimer’s, and it also is characterized by a more rapid onset, specifically of difficulty using or understanding language. This contrasts with Alzheimer’s disease, where most patients experience a more subtle and gradual decline that typically affects only memory. At Yale, Dr. Strittmatter and his colleagues are studying frontotemporal dementia at the molecular level in an attempt to understand what precipitates the degradation of those particular nerve cells. Previous research at other labs indicated that some forms of frontotemporal dementia are inherited, associated with a mutation in the gene for a protein called progranulin. Now, in lab and animal experiments, Dr. Strittmatter’s team has discovered that another protein called sortilin binds or interacts with progranulin, causing a decrease in the amount of progranulin that can be absorbed by cells in the brain, possibly contributing to the development of frontotemporal dementia. Both progranulin and sortilin are found throughout the body and, while it is not known exactly how they function in the brain, it is believed that they help protect the health of neurons.Dr. Strittmatter described these findings as the "first step" to understanding frontotemporal dementia, noting that they set the stage to begin working to find a therapeutic drug. "Eventually we hope to find a way to intervene in the disease and prevent or alleviate symptoms of this type of dementia," he said. Results of the study appeared in the November 18, 2010, issue of Neuron. Use It or Lose ItScientists have made significant progress over the last decade in distinguishing Alzheimer’s from other types of dementia, mainly by using positron emission tomography (PET) scans to detect amyloid plaques in the brain -- but at present, there is no effective drug to prevent or treat dementia, whatever its basis. Researchers continue to explore the different causes of dementia so that treatments can be tailored to the different forms of the disease, which promises to be more effective than coming up with a one-size-fits-all therapy. Meanwhile, for all of us, it is important to remain physically and intellectually active as we age. Use it or lose it, as they say. Keep walking or swimming, socializing with friends, visiting museums and playing challenging games such as chess or Scrabble. If you’d like to know more about frontotemporal dementia, you can find information from the Alzheimer’s Association at www.alz.org as well as from the more specialized Association of Frontotemporal Dementia (www.ftd-picks.org), where you can read about practical coping tips and join an online support group, message board or 24-hour helpline. Stephen M. Strittmatter, AB, MD, PhD, Vincent Coates Professor of Neurology, Yale School of Medicine, New Haven, Connecticut. Dr. Strittmatter is cofounder and director of the Yale Program in Cellular Neuroscience, Neurodegeneration and Repair.

dangerous side effect of certain antidepressants

How Antidepressants May Hurt Your ArteriesA number of weeks ago, I told you about a surprising and dangerous side effect of certain antidepressants -- they can mess up your heart’s rhythm (see the December 1, 2011 issue of Daily Health News). Today, I need to warn you about another little-known way that antidepressants may hurt your cardiovascular system -- they may thicken your artery walls. Few studies to date have examined this phenomenon, yet it poses a serious health risk. Narrowed arteries raise your risk for heart attack and stroke. Anyone taking an antidepressant should have this information. At Emory University School of Medicine in Atlanta, cardiologist Amit J. Shah, MD, and his colleagues decided to study this topic because the potential connection between antidepressant use and heart disease has been poorly understood. He presented his preliminary findings at the annual meeting of the American College of Cardiology this past spring in New Orleans. I gave him a call to get more details. one thickening, sickening problem To rule out genetics as a factor, Dr. Shah and his colleagues analyzed people from one of the largest twin databases in the country -- 513 pairs of identical and fraternal male twins (average age 55). At the time of the research, about 16% of the men were taking antidepressants -- all different kinds, though 60% of the antidepressants were selective serotonin reuptake inhibitors (SSRIs), such as fluoxetine (Prozac), duloxetine (Cymbalta) and sertraline (Zoloft). The researchers didn’t record how long the men had been taking the medications or the exact dosages. They used ultrasound to take one measurement of the thickness of the linings of the mens’ carotid arteries (the main arteries in the neck that supply blood to the brain). What the study found: As we grow older, our arteries naturally thicken at the rate of about 10 microns (one millionth of a meter) per year, but Dr. Shah discovered that participants who had been taking any type of antidepressant had experienced substantially greater artery thickening. When the researchers looked at the 59 twin pairs in which one twin was taking an antidepressant and the other wasn’t, the carotid artery lining was, on average, about 40 microns (about 5%) thicker in the twin taking the drug. The men were not followed to see who developed cardiovascular disease and who didn’t, but previous research has revealed that each 10-micron increase in carotid artery thickness is associated with a 1.8% increase in risk for cardiovascular disease, so that 40-micron increase in thickness could correlate to about a 7.2% increased risk for cardiovascular disease in the men who were on antidepressants. Another way to look at it: Their arteries were, in effect, four years older than their brothers’ arteries, noted Dr. Shah. This finding took into consideration other factors that can affect artery thickness, including alcohol and coffee intake, previous history of heart disease, history of post-traumatic stress disorder and depression. "A higher level of depressive symptoms was associated with higher artery thickness only in those taking antidepressants," said Dr. Shah. "Therefore, antidepressants may act synergistically with depressive symptoms to increase risk for artery thickness." WHY THE DISHEARTENING EFFECT?Dr. Shah theorized why the antidepressants might have been associated with having thicker arteries. Antidepressants, of course, work by increasing levels of neurotransmitters. Yet while these chemicals act in your brain to relieve symptoms of depression, Dr. Shah said that they also may cause blood vessels elsewhere in the body to constrict or tighten, which can lead to thicker arteries. Dr. Shah cautioned that this is an observational study -- not a test of cause-and-effect -- so we can’t definitively conclude from it that antidepressants cause artery thickening. Further research is needed to confirm these results. WEIGH THE PROS AND CONS So if you’re on an antidepressant, what should you do? "No one taking antidepressants should stop taking them based solely on the results of this study," said Dr. Shah. That is undoubtedly true. But keep in mind that, like all drugs, antidepressants have side effects -- some that we can see and feel (e.g., agitation, insomnia, sexual dysfunction) and perhaps others that we cannot. And if you’re considering taking an antidepressant, consider natural treatment first, such as exercise, healthy eating and talk therapy. Sit down with your doctor and carefully discuss the pros and cons for your particular situation. Source: Amit J. Shah, MD, cardiology fellow, Emory University School of Medicine, Atlanta.

Tuesday, January 3, 2012

Glaucoma: What Every Patient Should Know

Easy Trick That May Prevent GlaucomaAs you get older, you may be OK with the fact that your vision just isn’t what it used to be. But losing sight altogether is something that nobody -- myself included -- ever wants to imagine. That’s why I was pleased when I heard that there may be a simple new way to prevent glaucoma. Because it’s something that anybody can do -- exercise! The new study, published in the October issue of Investigative Ophthalmology & Visual Science, came from University College London Institute of Ophthalmology in England. To learn more, I called an expert who carefully examined the study, Harry A. Quigley, MD, an ophthalmologist, the director of the Glaucoma Center of Excellence at the Wilmer Eye Institute at Johns Hopkins University School of Medicine in Baltimore and author of Glaucoma: What Every Patient Should Know. EYEING PREVENTIONBefore we jumped into the research, Dr. Quigley gave me some background about how glaucoma develops. In glaucoma, there is slow, progressive damage to the optic nerve that can gradually lead to blindness if not treated. About 90% of glaucoma cases, he told me, are called open-angle. The scariest part about open-angle glaucoma is that there are no symptoms until irreversible damage happens, so if the person doesn’t get regular eye exams, then he won’t realize that he has glaucoma until blindness begins to set in. Some people who develop glaucoma (but not all) have what’s called high intraocular pressure (IOP), which is pressure in the eye. This new study, Dr. Quigley told me, focused on a measurement of something called ophthalmic perfusion pressure (OPP), which is the difference between your blood pressure and your IOP. So if your IOP is low, as you want it to be, then your OPP is higher (better). That means that your eyes are probably receiving more nourishing blood. But when your OPP is low, it means that circulation to and in the eyes is slowing -- which could raise your risk for glaucoma or worsen existing glaucoma. Keep in mind, said Dr. Quigley, that you can have a low OPP from either higher-than-normal IOP or lower-than-normal blood pressure (or both). Researchers investigated the relationship between physical activity and OPP. They looked at self-reported information from 5,650 adult men and women from about 15 years ago. Participants were grouped into one of two categories -- "active" or "less active." Researchers cross-referenced each participant’s level of physical activity with a measurement of OPP that was taken from the same people between 2006 and 2010. Results: Participants who had been "active" in the past had a 25% lower risk of having low OPP -- suggesting that they also had a lower risk of later developing glaucoma. What is especially uplifting about this discovery is that unlike taking drugs or having surgery, there is little risk involved in being active and exercising -- and it provides many other benefits that are well-documented! IMPROVED CIRCULATION = IMPROVED EYE HEALTHNow, of course, we all already know that exercise is, well, out of sight, but I found it intriguing that just someone’s general level of activity, as opposed to some fancy specific eye exercises, can have such a pronounced effect on your eye health. Dr. Quigley noted that exercise improves overall circulation, which brings better blood flow everywhere, including to the eyes. And, he added, this doesn’t mean that you have to hit the gym for vigorous workouts -- moderate activity, such as brisk walking that raises your heart rate for 20 minutes, is sufficient as long as you do it most days of the week.Besides moving around more, don’t forget to see an eye doctor regularly. Dr. Quigley advises everyone to start getting exams from an ophthalmologist (a medical doctor who can provide the full spectrum of eye care) at age 40, and depending on what your doctor advises, probably at least every one to two years after that. When you reach age 60, he said, you should get an eye exam annually, because age is a risk factor for glaucoma. And, he added, "It’s especially critical for those with a family history of glaucoma, those who are of certain ethnic origins (African American, Irish, Russian, Japanese, Hispanic, Inuit and Scandinavian) and/or those who are severely nearsighted, because these are also risk factors." Source(s): Harry A. Quigley, MD, director of the Glaucoma Center of Excellence at the Wilmer Eye Institute at Johns Hopkins University School of Medicine, Baltimore.

Monday, January 2, 2012

(( و إذا سألك عبادي عني فإني قريب أجيب دعوة الداع إذا دعان ))


بسم الله الرحمن الرحيممن روائع البيان في قوله تعالى (( و إذا سألك عبادي عني فإني قريب أجيب دعوة الداع إذا دعان )) ذكر علماء اللغة و البيان عنها ما يلي: 1- أنها الآية الوحيدة التي خالفت بقية الآيات التي تبدأ بسؤال الناس للنبي الكريم ، حيث كلها تأتي بصيغة ((يسألونك)) مثل ((يسألونك عن الشهر الحرام قتال فيه قل .. يسألونك عن الخمر و الميسر قل ...، يسألونك عن الأنفال قل ... ، و يسألونك عن اليتامى قل ... ، يسألونك ماذا أحل لهم قل ... ، و يسألونك ماذا ينفقون قل ... ، يسألونك عن الساعة أيان مرساها قل ... ، و يسألونك عن الروح قل ... ، و يسألونك عن الجبال فقل ... )) إلا هذه الآية ! فم ن عظمة الله أنه سبق المؤمنين بالسؤال و هم لم يسألوا بعد! و كأنه سؤال افتراضي ، فإن الله هو الذي وضع السؤال و بادر بالإجابة من قبل أن يُسأل حباً منه بالدعاء و بسرعة الإجابة ! فانظر إلى واسع رحمته! 2- على غرار (( و يسألونك عن الجبال فقل ينسفها ربي نسفا )) كان القياس أن يقول (و إذا سألك عبادي عني فقل ربي قريب يجيب دعوة الداع ) لكنه تبارك و تعالى تكفل بالإجابة بنفسه وقال (( فإني قريب أجيب دعوة الداع )) فابتدأ جوابه بأنه قريب للدلالة على عدم حاجته للوسطاء و الأولياء أولاً ، وللدلالة على حفاوته بالدعاء و بالسائلين ثانياً. فلم يتحدث بضمير الغائب عن ذاته فلم يقل ((يجيب دعوة الداع)) لأنه يدل على البعد و العلو ، بل نسبها لنفسه للدلالة على دنوه و قربه من السائلين ! 3- أنه تعالى لم يعلق الإجابة بالمشيئة كأن يقول (أجيبه إن أشاء) ، بل قطع و أكد بأنه يجيب دعوة الداع. 4- أنه قدم جواب الشرط على فعل الشرط ، فلم يقل (إذا دعان أستجب له) و ذلك للدلالة على قوة الإجابة و سرعتها. 5- أنه قال ((أجيب دعوة الداع إذا دعان)) و لم يقل (أجيب دعوة الداع إن دعان) و في هذا معانٍ بلاغية غاية في الدقة، منها أنه استخدم أداة الشرط ((إذا)) و لم يستخدم أداة الشرط ((إن)) ، فما الفرق بينهما؟السبب أن (إن) تستخدم للأحداث المتباعدة و المحتملة الوقوع و المشكوك فيها و النادرة و المستحيلة ، كقوله ((قل إن كان للرحمن ولد فأنا أول العابدين)) و قوله ((و إن طائفتان من المؤمنين اقتتلوا)) لأن الأصل عدم اقتتال المؤمنين ، و قوله ((ولكن انظر إلى الجبل فإن استقر مكانه فسوف تراني)) ، و لم يقل (إذا) استقر مكانه و قد علمنا أن الجبل دك دكاً! و كقوله ((قل أرأيتم إن جعل الله عليكم الليل سرمدا)). بينما (إذا) تعني المضمون حصوله أو كثير الوقوع ، مثل قوله ((كتب عليكم إذا حضر أحدكم الموت)) لأن الموت واقع لا محالة ! و قوله ((و ترى الشمس إذا طلعت تزاور عن كهفهم)) و قوله ((فإذا انسلخ الأشهر الحرم)) و قوله ((فإذا قضيت الصلاة)) ، و لذلك نرى أن كل أحداث يوم القيامة تأتي ب (إذا) و لم تأت بـ (إن) ، مثال ذلك قوله ((إذا زلزلت الأرض زلزالها)) و قوله ((إذا الشمس كورت و إذا النجوم انكدرت و إذا الجبال سيرت ...)) و قوله ((إذا وقعت الواقعة)) و غيرها من أحدث يوم القيامة حيث لم تأت أيا ًمنها بأداة الشرط (إن) لأنها تحتمل الندرة و عدم الوقوع. و من روعة هذا البيان هو حينما تأتيان معاً في موضع واحد فيستخدم (إذا) للكثرة و (إن للندرة) مثل قوله تعالى ((إذا قمتم إلى الصلاة فاغسلوا وجوهكم .. و إن كنتم جنبا )) فجاء بأ (إذا) للوضوء لأنه كثير الوقوع و (إن) للجنب لأنه نادر الحصول ، و مثل قوله ((فإذا أحصن فإن أتين بفاحشةٍ)) فالإحصان متكرر و الفاحشة من النوادر!فمن هذا نفهم أن المعنى من قوله تعالى ((إذا دعانِ)) أنه يشير إلى كثرة الدعاء و بأنه دعاء متكرر مستمر كثير و ليس نادراً قليلاً ! لأن الله يغضب إن لم يدعَ ، و القلب الذي لا يدعو قلبٌ قاسٍ ، ألم تر إلى قوله تعالى ((فأخذناهم بالبأساء و الضراء لعلهم يضرعون ، فلولا إذ جاءهم باسنا تضرعوا ولكن قست قلوبهم)) و قوله ((و لقد أخذناهم بالعذاب فما استكانوا لربهم و ما يتضرعون)). 6- ثم لاحظ أنه قال ((أجيب دعوة الداع)) و لم يقل ((أجيب الداع)) ! لأن الدعوة هي المستجابة و ليس شخص الداع ، و في هذا إشارة دقيقة جداً إلى مكانة الدعوة بغض النظر عن شخصية الداع! 7- قال ((عبادي)) بالياء و لم يقل ((عبادِ)) فما الفرق؟ ((عبادي)) تشير إلى عدد أكبر من ((عباد)) فالياء تعني أن مجموعة العباد أكثر ، أي يجيبهم كلهم على اختلاف ايمانهم و تقواهم ، كقوله تعالى للدلالة على الكثرة ((قل يا عبادي الذين أسرفوا على أنفسهم)) و المسرفون كثر ، و كقوله ((قل لعبادي يقولوا التي هي أحسن)) لأن أكثرهم يجادل ، أما للقلة فيقول ((فبشر عباد الذين يستمعون القول فيتبعون أحسنه)) و هؤلاء قلة ، و قوله ((وقل يا عباد الذين آمنوا اتقوا ربكم)) و المتقون قلة ! 8- لاحظ أنه قال : ((أجيب دعوة الداع)) و كان القياس أن يقول (أجيب دعوتهم)! و ذلك للدلالة على أنه يجيب دعوة كل داع و ليس فقط دعوة السائلين ، فوسع دائرة الدعوة و لم يقصرها على السائلين. 9- قال ((فإني قريب)) و لم يقل (أنا قريب) و هذا توكيد بـ (إن) المشددة للتوكيد ، لأن أنا غير مؤكدة. 10 - أن الآية توسطت آيات الصوم ، وهذا يعني أن الدعاء يستحب للصائم و أن للصائم دعوة لا ترد كما ورد في الأثر ( ما لم تكن بقطيعة رحم). الدعاء شعار الصائمين ، ومن عظمة الدعاء و منزلته عند الله أن الله أحاطه بآيات الصوم الذي قال عنه في الحديث القدسي (الصوم لي و أنا أجزي به) لأن الصوم من شعائر الإخلاص لله لأنه شَعيرة غير ظاهرة الأثر على صاحبها ما لم يرائي ، فكذا الدعاء أراده الله أن يكون خالصاً له و هو الذي يجزي به من دون شرك فيه لأحد ، من دون واسطة نبي أو ولي. اللهم صلِ وسلّم على الحبيب المصطفى و آله وأصحابه وأزواجه أجمعين